Metabolic alterations associated with T2D are well characterised by epidemiological and research based experiments. The pathogenesis and development of T2D is ascribed to four mechanisms; amplified Sophisticated glycation close merchandise (AGE) development, improved polyol pathway flux, activation of protein kinase C (PKC) isoforms, and greater hexosamine pathway flux[7]. Till just https://www.directivepublications.org/journal-of-diabetology-research/